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Highlights Aldosterone is the major mineralocorticoid produced by the adrenal zona glomerulosa. Glomerulosa cells are primarily regulated by angiotensin II, serum potassium and ACTH. Aldosterone synthesis involves two key phases, acute and chronic. Cholesterol delivery into mitochondria is the rate-limiting step in the acute phase. Aldosterone synthase (CYP11B2) is the rate-limiting step in.
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OlGly and OlAla suppress EtOH self-administration and preference. We have previously shown that OlGly was elevated in a region-speci c manner in the NAc of male mice chronically treated with fi.
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Aldosterone is a major mineralocorticoid steroid hormone secreted by glomerulosa cells in the adrenal cortex. It regulates a variety of physiological responses including those to oxidative stress, inflammation, fluid disruption, and abnormal blood pressure through its actions on various tissues including the kidney, heart, and the central nervous system. Aldosterone synthesis is primarily.
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Rationale The fatty acid amide oleoyl glycine (OlGly) and its more stable methylated form oleoyl alanine (OlAla) reduce naloxone-precipitated morphine withdrawal (MWD)-induced conditioned gaping (nausea) responses in rats. In addition, OlGly has been shown to reduce lithium chloride (LiCl)-induced conditioned gaping in rats and vomiting in Suncus murinus (house musk shrews). Objectives Here.
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The endogenous amide N-Oleoylglycine (OlGly) and its analog N-Oleoylalanine (OlAla), have been shown to interfere with the affective and somatic responses to acute naloxone-precipitated MWD in male rats. Here we evaluated the potential of a single dose (5 mg/kg, ip) which alleviates withdrawal of these endogenous fatty acid amides to modify tolerance to anti-nociception, hyperthermia, and.
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ATP1A1 mutations disrupt this function and cause cell membrane depolarization and cellular acidification because of a pathological H + leak, resulting in aldosterone overproduction. 55,61,69 The ATP2B3 gene encodes the plasma membrane Ca 2+ ATPase isoform 3, which regulates intracellular Ca 2+ homeostasis by transporting cytoplasmic Ca 2+ out of the cells.
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Coelution of OlAla with an isobaric compound (shown separated in Figure 2 with the presented mobile phase gradient) occurred when the starting mobile phase was 60% mobile phase B which did not allow an isobaric compound of OlAla to chromatographically resolve. Using the presented method allowed for the separation of potential interferences from isobaric compounds found in the brain tissue samples.
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Since in EtOH self-administered mice the expression of OlAla significantly elevated in the principle areas of the reward system (PFC, NAc, and VTA, Fig. 2B and Table 1), we hypothesized that OlAla.
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Primary aldosteronism (PA) is one of the most frequent curable forms of secondary hypertension. It can be caused by the overproduction of aldosterone in one or both adrenal glands. The most common subtypes of PA are unilateral aldosterone over-production due to aldosterone-producing adenomas (APA) or bilateral aldosterone over-production due to bilateral hyperaldosteronism (BHA). Utilizing the.